Temporal and spatial expression of matrix metalloproteinases and tissue inhibitors of metalloproteinases following myocardial infarction
Cardiac wound healing post-myocardial infarction: a novel method to target extracellular matrix remodeling in the left ventricle
Extracellular matrix turnover and signaling during cardiac remodeling following MI: causes and consequences
In vivo matrix metalloproteinase-7 substrates identified in the left ventricle post-myocardial infarction using proteomics
Tumor necrosis factor-alpha–converting enzyme roles in hypertension-induced hypertrophy: look both ways when crossing the street
Stem cell mechanisms during left ventricular remodeling post-myocardial infarction: Repair and regeneration
C-terminal fragment of transforming growth factor beta-induced protein (TGFBIp) is required for apoptosis in human osteosarcoma cells
A Novel Collagen Matricryptin Reduces Left Ventricular Dilation Post-Myocardial Infarction by Promoting Scar Formation and Angiogenesis
CC chemokine receptor 5 deletion impairs macrophage activation and induces adverse remodeling following myocardial infarction
Transgenic overexpression of matrix metalloproteinase-9 in macrophages attenuates the inflammatory response and improves left ventricular function post-myocardial infarction
AMP activated protein kinase 2 protection during hypertension-induced hypertrophy: a common mediator in the signaling crossroads
